Citation Impact 2023
Journal Impact Factor: 6.1
5-year Journal Impact Factor: 7.1
Source Normalized Impact per Paper (SNIP): 1.865
SCImago Journal Rank (SJR): 2.578
Speed 2023
Submission to first editorial decision (median days): 14
Submission to acceptance (median days): 129
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Articles
Page 106 of 108
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Citation: Breast Cancer Research 2000 2(Suppl 1):S.35
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Clinical trials in breast cancer; do we have the right approaches?
Citation: Breast Cancer Research 2000 2(Suppl 1):S.33 -
Hypoxia and tumour angiogenesis involved in breast cancer progression and targets for therapy
Citation: Breast Cancer Research 2000 2(Suppl 1):S.28 -
p53 action in apoptosis and senescence
Citation: Breast Cancer Research 2000 2(Suppl 1):S.22 -
p53-mediated apoptosis and genomic instability syndromes
Citation: Breast Cancer Research 2000 2(Suppl 1):S.20 -
Drug discovery in the p53 pathway
Citation: Breast Cancer Research 2000 2(Suppl 1):S.18 -
Characterisation of micrometastatic tumor cells
Citation: Breast Cancer Research 2000 2(Suppl 1):S.16 -
Clinical translation of progress in molecular endocrinology
Citation: Breast Cancer Research 2000 2(Suppl 1):S.12 -
Estrogen receptors α and β in the rodent mammary gland
Citation: Breast Cancer Research 2000 2(Suppl 1):S.11 -
Comparative genomic hybridisation analysis of myoepithelial carcinoma of the breast
Citation: Breast Cancer Research 2000 2(Suppl 1):P8.06 -
Relation of gene expression-based tumor subclasses to clinical phenotypes
Citation: Breast Cancer Research 2000 2(Suppl 1):P8.11 -
Screening for BRCA1 gene mutations by capillary electrophoretic REF-SSCP
Citation: Breast Cancer Research 2000 2(Suppl 1):P8.04 -
REAL Real-Time!
Citation: Breast Cancer Research 2000 2(Suppl 1):P8.02 -
The role of coactivators in oestrogen action
Citation: Breast Cancer Research 2000 2(Suppl 1):S.08 -
Identification of two novel breast cancer associated genes by the differential display method
Citation: Breast Cancer Research 2000 2(Suppl 1):P8.08 -
Cytokeratin and mammaglobin as tumor markers in patients with high risk breast cancer
Citation: Breast Cancer Research 2000 2(Suppl 1):P7.05 -
AKT-1 and BCL-2 co-expression in breast cancer patients correlates with better survival
Citation: Breast Cancer Research 2000 2(Suppl 1):P7.02 -
Ribozymes targeting the tumor suppressor BRCA1 lead to increased chemosensitivity to cisplatin and etoposide and chemoresistance to microtubule-interfering agents in HBL100 breast cancer cell line
Citation: Breast Cancer Research 2000 2(Suppl 1):P7.04 -
Mammary epithelial cells immortalized by over-expression of the catalytic subunit of human telomerase may represent a model for mammary epithelial differentiation
Citation: Breast Cancer Research 2000 2(Suppl 1):P6.03 -
Setting up an efficient and reliable screening method for novel c-Jun N-terminal kinase substrates
Citation: Breast Cancer Research 2000 2(Suppl 1):P5.05 -
The pathology of inherited breast tumours
Citation: Breast Cancer Research 2000 2(Suppl 1):S.05 -
p16INK4A as a predictive factor in patients with locally advanced breast cancer treated with neoadjuvant doxorubicin monotherapy
Citation: Breast Cancer Research 2000 2(Suppl 1):P5.01 -
Estrogen-dependent c-jun upregulation may control cyclin D1 expression
Citation: Breast Cancer Research 2000 2(Suppl 1):P5.03 -
Studies of TP53 haplotypes in relation to LOH and TP53 mutations in breast cancer patients
Citation: Breast Cancer Research 2000 2(Suppl 1):P4.06 -
Analysis of Fhit expression in stages of breast cancer progression
Citation: Breast Cancer Research 2000 2(Suppl 1):P4.12 -
The GSTT1 genotype is associated with p53 mutation status of breast cancer tumours
Citation: Breast Cancer Research 2000 2(Suppl 1):P4.05 -
BRCA2 interacting proteins
Citation: Breast Cancer Research 2000 2(Suppl 1):P4.02 -
Expression of variant forms of the MUC1 gene correlates with the invasiveness of breast cancer cells
Citation: Breast Cancer Research 2000 2(Suppl 1):P4.16 -
Expression of Akt kinases related to ErbB2 and results of adjuvant therapy of breast cancer
Citation: Breast Cancer Research 2000 2(Suppl 1):P3.04 -
HER-2 activation and TP53 inactivation as predictors of survival in breast cancer
Citation: Breast Cancer Research 2000 2(Suppl 1):P3.07 -
Activation of the Her2/neu receptor leads to antiestrogen resistance, but acquired resistance is not caused by altered ErbB expression and activation
Citation: Breast Cancer Research 2000 2(Suppl 1):P3.03 -
Phosphorylation of human progesterone receptors by MAPKs mediates transcriptional synergy between progestins and MEKK1 in human breast cancer cells
Citation: Breast Cancer Research 2000 2(Suppl 1):P2.06 -
Germline mutations of BRCA1-associated RING domain (BARD1) gene in breast and/or ovarian families negative for BRCA1 and 2 alterations
Citation: Breast Cancer Research 2000 2(Suppl 1):P1.15 -
Constitutional alterations of 9p23-24 in independent BRCA2 breast cancer families
Citation: Breast Cancer Research 2000 2(Suppl 1):P1.13 -
The importance of breast cancer research from a patient's view: the voices and visions of advocates
Citation: Breast Cancer Research 2000 2(Suppl 1):S.01 -
Germline TP53mutations in Finnish breast cancer patients
Citation: Breast Cancer Research 2000 2(Suppl 1):P1.09 -
Altered expression of estrogen receptor-α variant messenger RNAs between adjacent normal breast and breast tumor tissues
Using semiquantitative reverse transcription-polymerase chain reaction assays, we investigated the expression of variant messenger RNAs relative to wild-type estrogen receptor (ER)-α messenger RNA in normal br...
Citation: Breast Cancer Research 2000 2:64 -
Smoking and high-risk mammographic parenchymal patterns: a case-control study
Current smoking was strongly and inversely associated with high-risk patterns, after adjustment for concomitant risk factors. Relative to never smokers, current smokers were significantly less likely to have a...
Citation: Breast Cancer Research 2000 2:59 -
Gene therapy for carcinoma of the breast: Genetic ablation strategies
The gene therapy strategy of mutation compensation is designed to rectify the molecular lesions that are etiologic for neoplastic transformation. For dominant oncogenes, such approaches involve the functional ...
Citation: Breast Cancer Research 2000 2:45 -
Gene therapy for carcinoma of the breast: Therapeutic genetic correction strategies
Gene therapy is a therapeutic approach that is designed to correct specific molecular defects that contribute to the cause or progression of cancer. Genes that are mutated or deleted in cancers include the can...
Citation: Breast Cancer Research 2000 2:28 -
Gene therapy for carcinoma of the breast: Genetic toxins
Gene therapy was initially envisaged as a potential treatment for genetically inherited, monogenic disorders. The applications of gene therapy have now become wider, however, and include cardiovascular disease...
Citation: Breast Cancer Research 2000 2:22 -
Gene therapy for carcinoma of the breast: Genetic immunotherapy
Advances in gene transfer technology have greatly expanded the opportunities for developing immunotherapy strategies for breast carcinoma. Genetic immunotherapy approaches include the transfer of genes encodin...
Citation: Breast Cancer Research 2000 2:15 -
Beyond the genome to tissue proteomics
Citation: Breast Cancer Research 2000 2:13 -
Breast cancer prevention
Citation: Breast Cancer Research 2000 2:10 -
Microarray foray
Citation: Breast Cancer Research 2000 2:8 -
Modeling Mammary Cancer in Mice Conference, Jackson Laboratory, Bar Harbor, Maine, 5-8 October, 1999
Citation: Breast Cancer Research 2000 2:E003 -
Mouse models for breast cancer
Citation: Breast Cancer Research 2000 2:2 -
6th Nottingham International Breast Cancer Conference (22-24 September 1999)
Citation: Breast Cancer Research 2000 2:E002 -
International Conference on the Biology of the Mammary Gland. Tours, France (16-18 September 1999)
Citation: Breast Cancer Research 2000 2:E001 -
Breast Cancer Research non-peer-review research depository
Citation: Breast Cancer Research 2000 2:1